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About me

Cameron Jones is a microbiologist/mycologist and former University academic. He has published over 80 peer reviewed papers and conference proceedings and over 3500+ technical reports on air quality and microbial pathogens in the built environment. His PhD was in fungi. He is also the Editor-In-Chief for the Journal of Bacteriology & Mycology: Open Access. He operates Biological Health Services which is an environmental microbiology lab and consultancy and is an adjunct academic at the National Institute of Integrative Medicine. 

What we breathe is fundamental to our health. Air pollution kills an estimated seven million people worldwide every year. The World Health Organization has data showing that 9 out of 10 people breathe air containing high levels of contaminants. In many cases these contaminants are biological in origin and include mould spores, bacteria, yeasts, pollens, parasites and virus. The microbial world is diverse and not always pathogenic in terms of direct disease. However, in the air-stream, these cells easily fragment over time and generate micron and sub-micron dusts that are inhaled and become deposited inside our homes. My role is to show how science and health are connected and that despite climate change and urbanisation, there is hope for wellness. I look forward to you joining with me

BSc, Grad Dip App Sci, PhD

The Morning Show, Ch 7.

28th May 2024.

Winter mould with Dr Cameron Jones

Dr. Cameron Jones is recognized for his expertise in public health matters, providing valuable insights on TV appearances related to mold and indoor air quality. His engaging discussions resonate with audiences and media professionals alike, making him a sought-after expert for various shows.

TV Appearances Showcase

Dirty beauty

DIRTY BEAUTY 💄 Dr Cameron Jones reveals what's lurking in your make-up brushes, and unhygienic habits to break 🧐 #TMS7 – Straight after Sunrise, weekdays on Channel 7 and 7Plus

Posted by The Morning Show on Wednesday 7 August 2024

The Morning Show, Ch 7.

8th August 2024

Dr Cameron Jones reveals what's lurking in your make-up brushes, and unhygienic habits to break

Just how dirty is your Stanley cup?

Just how dirty is your Stanley cup? Reusable bottles can have 40,000 times more bacteria than toilet seats. It's time to talk about why your favourite fashion statement is harbouring a heap of bacteria 👀 #TMS7 – Straight after Sunrise, weekdays on Channel 7 and 7plus

Posted by The Morning Show on Thursday 8 February 2024

The Morning Show, Ch 7

9th February 2024

Just how dirty is your Stanley cup?

The Morning Show

1st December 2023

Microorganisms on Bras: What Happens When You Don't Wash Them Often?

YouTube

Without medical breakthroughs, more than 6.4M Australians will be diagnosed with dementia in the next 40 years at a cost of more than $1 trillion. This week I want to discuss several exciting publications that have appeared in the research literature in 2020. It’s not just dementia, other diseases like Alzheimer’s, multiple sclerosis, Parkinson’s, stroke, and even depression are increasingly being linked to something called mitochondrial dysfunction. The literature shows that 2 approaches called: methylene blue and photobiomodulation might be very effective. But why? This issue of mitochondria and its’ link with inflammatory processes has been known for some time. Especially, with respect to how fungal pathogens and environmental fungi seem to induce similar mitochondrial dysfunction in humans. I will look at what’s known about this and then review a paper from 2020 showing that fungi have been found in the brains of people with Parkinson’s disease. This has been termed ‘polymicrobial invasion’ and suggests that some brain disorders are catalyzed by infections. The main paper I want to focus on as part of this Livestream came out on August 14th, 2020, and shows that brain inflammation, cytokine expression, and memory can be actively modulated with a common anti-fungal drug. This is breakthrough research and suggests this treatment could offer real relief in the near term. Join me on the Livestream or on the Podcast to find out which drug offers this potential microbiological benefit and what evidence supports this claim. REFERENCES: Mudarri D, Fisk WJ. Public health and economic impact of dampness and mold [published correction appears in Indoor Air. 2007 Aug;17(4):334]. Indoor Air. 2007;17(3):226-235. doi:10.1111/j.1600-0668.2007.00474.x Yeo, I.J., Yun, J., Son, D.J. et al. Antifungal drug miconazole ameliorated memory deficits in a mouse model of LPS-induced memory loss through targeting iNOS. Cell Death Dis 11, 623 (2020). https://doi.org/10.1038/s41419-020-2619-5 Pisa D, Alonso R, Carrasco L. Parkinson's Disease: A Comprehensive Analysis of Fungi and Bacteria in Brain Tissue. Int J Biol Sci 2020; 16(7):1135-1152. doi:10.7150/ijbs.42257. Available from http://www.ijbs.com/v16p1135.htm Yang L, Youngblood H, Wu C, Zhang Q. Mitochondria as a target for neuroprotection: role of methylene blue and photobiomodulation. Transl Neurodegener. 2020;9(1):19. Published 2020 Jun 1. doi:10.1186/s40035-020-00197-z GBD 2016 Neurology Collaborators. Global, regional, and national burden of neurological disorders, 1990-2016: a systematic analysis for the Global Burden of Disease Study 2016. Lancet Neurol. 2019;18(5):459-480. doi:10.1016/S1474-4422(18)30499-X Hamblin MR. Shining light on the head: Photobiomodulation for brain disorders. BBA Clin. 2016;6:113-124. Published 2016 Oct 1. doi:10.1016/j.bbacli.2016.09.002 Fisher MC, Gurr SJ, Cuomo CA, et al. Threats Posed by the Fungal Kingdom to Humans, Wildlife, and Agriculture. mBio. 2020;11(3):e00449-20. Published 2020 May 5. doi:10.1128/mBio.00449-20 Zhang JM, An J. Cytokines, inflammation, and pain. Int Anesthesiol Clin. 2007;45(2):27-37. doi:10.1097/AIA.0b013e318034194e #miconazole #dementia #inflammation
How an common anti-fungal drug could help stop brain disease
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Mast Cell Activation Syndrome (MCAS) is a very serious, often overlooked condition. About one in four people with the disease cannot work or participate in normal day-to-day activities. It is a disease that affects people of all ages or gender, but one funny thing about it is that it is often unrecognized and possibly remains undiagnosed in many cases. MCAS is a group of diseases involving the immune system. When the triggers occur, antigens are raised inside the body, which might result in itchiness, for example. But this also leads to a histamine cascade, which causes contraction of the respiratory vessels. Then blood vessels dilate and result in gastric acid secretion. Many people with MCAS can have very serious life-threatening reactions such as anaphylaxis. Typical symptoms include itching, headaches, brain fog, fatigue, heart palpitations, hives, shortness of breath, gastrointestinal problems, wheezing, low blood pressure, anxiety, and, very typically, flushing or swelling, which appears on the face or body. According to the American Academy of Allergy Asthma & Immunology, Mast Cell Activation Syndrome is a condition in which the patient experiences repeated episodes of the symptoms of anaphylaxis – allergic symptoms such as hives, swelling, low blood pressure, difficulty breathing, and severe diarrhea. High levels of mast cell mediators are released during those episodes. For these reasons, it is therefore important that you are very much aware of the top seven underlying factors that can contribute to Mast Cell Activation Syndrome and avoid them completely so you can live a healthier, happier life. READ MORE ON THE BLOG HERE: https://www.drcameronjones.com/blog/top-7-mast-cell-activation-syndrome-triggers-you-should-avoid #mastcell #MCAS #mastcellactivationsyndrome
Top 7 Mast Cell Activation Syndrome Triggers You Should Avoid
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What are the scientists saying about how you might be able to treat yourself to minimise the risk of infection from the SARS-CoV-2 virus? The papers I want to review in todays Livestream demonstrate that oral antiseptics like povidine-iodine are effective at inactivating the coronavirus. The effect works at a variety of concentrations after 60 seconds contact time. The research papers show this works as oral and nasal rinses. This is an important show - since these papers might help you or a loved one. Of course, speak to your medical doctor or other health care practitioner - but from an infection control perspective, these research results are very persuasive. Use masks and consider adopting the scientific results detailed in these papers into your own harm minimisation strategy during the pandemic. This is not an alternative to testing nor accessing your medical doctor if you have any symptoms that could be Covid-related, but this common antiseptic could help and that’s the point of today’s Livestream. REFERENCES: 1. In Vitro Efficacy of Povidone-Iodine Nasal And Oral Antiseptic Preparations Against Severe Acute Respiratory Syndrome-Coronavirus 2 (SARS-CoV-2).Jesse Pelletier, Belachew Tessema, Jonna Westover, Samantha Frank, Seth Brown, Joseph Capriotti. medRxiv 2020.05.25.20110239; doi: https://doi.org/10.1101/2020.05.25.20110239. (MAY 26th 2020) 2. Khan MM, Parab SR, Paranjape M. Repurposing 0.5% povidone iodine solution in otorhinolaryngology practice in Covid 19 pandemic [published online ahead of print, 2020 Jun 18]. Am J Otolaryngol. 2020;41(5):102618. doi:10.1016/j.amjoto.2020.102618 (JUNE 5TH 2020) 3. Anderson DE, Sivalingam V, Kang AEZ, et al. Povidone-Iodine Demonstrates Rapid In Vitro Virucidal Activity Against SARS-CoV-2, The Virus Causing COVID-19 Disease [published online ahead of print, 2020 Jul 8]. Infect Dis Ther. 2020;1-7. doi:10.1007/s40121-020-00316-3 (JUNE 2ND 2020) 4. Dev Kumar G, Mishra A, Dunn L, et al. Biocides and Novel Antimicrobial Agents for the Mitigation of Coronaviruses. Front Microbiol. 2020;11:1351. Published 2020 Jun 23. doi:10.3389/fmicb.2020.01351 (23RD JUNE 2020) 5. Contini C, Enrica Gallenga C, Neri G, Maritati M, Conti P. A new pharmacological approach based on remdesivir aerosolized administration on SARS-CoV-2 pulmonary inflammation: A possible and rational therapeutic application [published online ahead of print, 2020 May 24]. Med Hypotheses. 2020;144:109876. doi:10.1016/j.mehy.2020.109876 (22ND MAY 2020) 6. Funk CD, Laferrière C, Ardakani A. A Snapshot of the Global Race for Vaccines Targeting SARS-CoV-2 and the COVID-19 Pandemic. Front Pharmacol. 2020;11:937. Published 2020 Jun 19. doi:10.3389/fphar.2020.00937 #iodine #Betadine #covid19au 7. Potential Medicines to Treat COVID-9. https://www.safetyandquality.gov.au/sites/default/files/2020-05/Potential%20medicines%20to%20treat%20COVID-19%20%2815%20May%202020%29.pdf 8. Bigliardi, P. L., Alsagoff, S. A. L., El-Kafrawi, H. Y., Pyon, J.-K., Wa, C. T. C., & Villa, M. A. (2017). Povidone iodine in wound healing: A review of current concepts and practices. International Journal of Surgery, 44, 260–268. doi:10.1016/j.ijsu.2017.06.073
Could Iodine Nasal & Oral Antiseptics Help Protect You Against SARS-CoV-2?
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Sickness behaviour following mould exposure has finally been validated. It turns out that anxiety, memory deficits and pain thresholds are all switched on by BOTH toxic and non-toxic mould. The research shows that mould spores and mould cell wall fragments can adversely affect the brain and influence behaviour by challenging and activating the innate immune system. In this week's Livestream I'm going to be talking about these important findings and making them understandable for a lay audience. REFERENCES: Harding CF, Pytte CL, Page KG, Ryberg KJ, Normand E, Remigio GJ, DeStefano RA, Morris DB, Voronina J, Lopez A, Stalbow LA, Williams EP and Abreu O. Mold inhalation causes innate immune activation, neural, cognitive and emotional dysfunction. Brain, Behaviour, and Immunity. Available online 18th November 2019. https://www.sciencedirect.com/science/article/pii/S0889159119303010 Anand KS and Dhikav V. Hippocampus in health and disease: An overview. Ann Indian Acad Neurol. 2012 Oct-Dec; 15(4): 239–246. https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3548359/ Boato, F., Hechler, D., Rosenberger, K. et al. Interleukin-1 beta and neurotrophin-3 synergistically promote neurite growth in vitro . J Neuroinflammation 8, 183 (2011) doi:10.1186/1742-2094-8-183 Cui J, Chen Y, Wang HY, Wang RF. Mechanisms and pathways of innate immune activation and regulation in health and cancer. Hum Vaccin Immunother. 2014;10(11):3270–3285. doi:10.4161/21645515.2014.979640 Jones, CL. Environmental exposure patterns for airborne particulate matter (PM2.5 and PM10) inside residential properties. ACNEM Journal. 2016; 35(1): 20-21. Enjoy & 👇Comment Your Thoughts Below👇 ★ Follow & Like On Facebook: 👉 https://www.facebook.com/biologicalhealth ★ Follow BIOLOGICAL HEALTH SERVICES On Instagram: 👉 https://www.instagram.com/biologicalhealthservices ★ Website for BIOLOGICAL HEALTH SERVICES: 👉 https://www.biologicalhealthservices.com.au ★ Website for DR CAMERON JONES: 👉 https://www.drcameronjones.com ★ Subscribe On YouTube: 👉 http://www.youtube.com/drcameronjones 👉 http://www.drcameronjones.tv ★ Follow DR CAMERON JONES On Twitter: 👉https://www.twitter.com/drcameronjones ★ Follow DR CAMERON JONES On Instagram: 👉https://www.instagram.com/drcameronjones ★ Live Streaming Calendar: 👉https://www.drcameronjones.live #mould #anxiety #breakthrough
Breathing in mould spores causes anxiety, memory deficits and increases pain perception
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This video follows on from an OpEd I wrote for TrialSite News: https://trialsitenews.com/could-tucker-carlsons-nicotine-gum-habit-stop-covid/ Early in the pandemic researchers were scrambling to make sense of the data, much of this from China which has a high smoking prevalence. One review showed that current smokers were less likely to have an adverse COVID-19 outcome compared with former smokers. A groundbreaking pre-print from France in mid-2020 then examined if nicotine replacement therapy (NRT) given to smokers split into two groups (those with and without major smoking-related disease) would have an effect on hospitalization with COVID-19. In people without comorbidities from smoking-related diseases, nicotine replacement therapy was associated with a decreased risk of hospitalization. The ratio of NRT versus no NRT for patients hospitalized with COVID-19 was 151:496 while the ratio of death or intubation was 13:73. However, these trends did not carry over for patients having known smoking-related diseases. Continuing research then suggested that nicotine, not smoking, could offer some protection against COVID-19. There are even five clinical trials underway in France. They are investigating the potential protective effect of nicotine patches to prevent adverse outcomes like the need for mechanical ventilation or transfer to intensive care and even as a preventative. According to ClinicalTRials.gov these studies are active or recruiting or show as complete, but as yet, no new results have been published. Since the coronavirus pandemic began, doctors and scientists have observed that daily active cigarette smokers are less likely to become severely infected or hospitalized despite the fact the virus targets the lungs. Reformed puffers aren't so lucky and are more likely to become ill due to smoke damaged respiratory or cardiovascular systems, or because of immune compromise. Ultimately, COVID-19 is an inflammatory syndrome illness. Current thinking is that the principal receptor allowing the SARS-CoV-2 virus to infect cells is through the angiotensin-converting enzyme 2 (ACE2) found in the lungs, small intestine, colon, kidney, brain and heart. However, there is some doubt this is the only way the virus causes infection since high ACE2 levels may be beneficial rather than harmful, especially in people with lung injury, like smokers. When lungs are infected by bacteria, fungi or viruses a strong inflammatory response occurs. Illness severity depends on lung macrophages which switch between pro-inflammatory and anti-inflammatory modes by releasing high levels of inflammatory cytokines. This signals other cells to challenge the infection and helps with healing. Severe COVID-19 symptoms and mortality are related to an over-responsive immune system leading to the cytokine storm effect. What if other receptors or activators control whether the coronavirus can successfully gain entry into cells? This paradox is explained by the nicotine hypothesis where the addition of this alkyloid drug exerts its protective effect via the nicotinic acetylcholine receptor (nAChR) by modulating ACE2 expression and preventing a hyperinflammatory response. By increasing ACE2 expression in cells, lung injury and kidney damage can be limited. Nicotine may also help reduce platelet aggregation and stop clot formation since it has known antioxidant and anti-inflammatory properties. Although no published clinical results are available at this time to supplement the earlier work, a review of over forty international studies shows clear differences between smoking history and non-severe and severe COVID illness; with smokers being more than twice as likely to suffer severe illness. But there are numerous contradictory studies from China, South Korea and India showing protection from SARS-CoV-2 infection from smoking. I asked Professor Dautzenberg about the importance of therapeutics like nicotine compared with vaccines and their role in public health messaging. He is of the view that the "vaccine is much more active but it's important to understand everything about COVID-19 including the interaction of nicotine with COVID". So even though we'll have to wait for the results from these trials, we all want to know whether nicotine might work as a preventative or to limit hospitalisation or severity. To this, Prof. Dautzenberg commented "the only practical recommendation is to offer complete nicotine substitution in all hospitalised COVID" patients. Based on the scientific research alone, repurposing already available and approved nicotine products like gum or patches could help restore the compromised function of the nicotinic cholinergic system under coronavirus attack. This could be a valuable, low-cost therapeutic option for treating COVID-19 by reducing inflammation and suppressing the cytokine storm. #nicotine #nicotinehypothesis #coronavirus
Could Nicotine Stop Covid
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Science is all about observation and forming hypotheses to answer questions. For scientists like myself, I’m interested in how the environment affects health, and specifically, how water-damaged buildings and exposure to poor indoor air quality can affect quality of life. For this week on The Mould Show we're going to be talking about fungal mycotoxins and myalgic encephalomyelitis termed ME for short and chronic fatigue syndrome (ME/CFS). The reason I've chosen this topic is that I've met a family whom I've now done three separate mould inspections for. Why this is unusual is that the symptoms experienced by the female occupant and her daughter are classic ME/CFS symptoms. Across all 3 of the different properties hidden mould problems were discovered. That information in itself was useful for this family and helped them to break their lease due to the fact that there was verified mould present. However, that's not the point of this podcast and even the positive mould confirmation as an environmental trigger didn’t help the mother and daughter from understanding why they reacted so strongly when indoors. They thought they were going crazy, since how could 3 different properties cause them so much trouble? They couldn’t understand why some buildings caused no issues, and the 3 they’d chosen to rent all set them off? This has bothered me all year. I know mould is a huge issue for some people – but what exactly might be going on for THIS family? A very interesting article on Substack focuses on his experience with chronic pain and ME/CFS and how after 25 years of suffering, he discovered by chance, following a recommendation from a friend to read a book that talked about the connection between ME and chronic fatigue syndrome and mitochondrial dysfunction. So that is the true focus of the topic today. • Could exposure to fungal mycotoxins be the reason why they were tired and experiencing pain? • Could this be the reason you’re tired? Is this another aspect to environmental illness? • And could this be classified as Myalgic Encephalomyelitis or Chronic Fatigue Syndrome (ME/CFS)? • Are there any patient-specific tests to verify ME/CFS or mould exposure and what can be done to alleviate the symptoms? Firstly, let's define what ME/CFS is. There are three core symptoms + at least one or more other symptoms which include: 1. an inability to participate in routine activities that would have been possible before becoming ill and that lasts for greater than six months. 2. the fatigue which is also a primary symptom tends to worsen after physical, mental or emotional effort. 3. and does not resolve after sleep, termed ‘unrefreshing sleep’. Other problems include: a) an inability to concentrate or memory impairment and/or b) something called orthostatic intolerance. Which means that when people stand up, they feel lightheaded or dizzy and can even faint. The family that moved into those three different properties all complained that after a few days of occupying the mould contaminated building the wife especially experienced episodes of unexpected fainting and collapsing into her husband's arms. Those 3 inspections coupled with reading the Substack article led me to investigate further whether there was any published literature, focusing on exposure to fungal cells or their mycotoxins in the ME/CFS literature. Very recently a publication appeared in the International Journal of Environmental Research and Public Health that has looked at exactly this topic. These scientists looked at the levels of Aspergillus toxins like aflatoxin, ochratoxin, and gliotoxin in the urine of 236 ME/CFS patients who had a known exposure to mould from water damaged buildings. The evidence from two types of urine mycotoxin testing from Real Time Laboratories as well as from the Great Plains Laboratories revealed that 92.4% of ME/CFS patients who had exposure to water damaged buildings had mycotoxin evidence in their urine, with ochratoxin being the most prevalent. This study is extremely important because it validates what many of us mycologists and microbiologists have known for many years - that exposure to water damaged buildings leads to mould exposure and that it is the mycotoxins and not just the intact cell which contribute to adverse health. In this Livestream, I'll also be extending on the concept of mitochondrial dysfunction and we'll be looking at three additional publications which have looked at how the individual can improve their mitochondrial activity for better health. REFERENCES: Wu, T.Y.; Khorramshahi, T.; Taylor, L.A.; Bansal, N.S.; Rodriguez, B.; Rey, I.R. Prevalence of Aspergillus-Derived Mycotoxins (Ochratoxin, Aflatoxin, and Gliotoxin) and Their Distribution in the Urinalysis of ME/CFS Patients. Int. J. Environ. Res. Public Health 2022, 19, 2052. https://doi.org/10.3390/ijerph19042052
Fungal mycotoxins & ME/CFS could this be why you're tired?
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You might not know it but there is an increasing incidence of mould related illness worldwide due to changes in building practices and efforts to cut heating energy losses. This has led to building construction of homes that don't breathe as effectively, the use of lightweight construction practices, often using risky and poorly tested building materials. This combination of poor building practices and attention to detail, means that errors in the construction process often lead to a series of unwanted building defects that conspire to make water management problems turn into mould problems that are often hidden behind wall, floor or ceiling linings. These problems can lead to mould related disease and the defining hallmark of this is chronic exposure of the respiratory system to bioaerosols, like mould. In this presentation, I want to review something called salt therapy, which is considered a complementary method for the treatment of respiratory tract illnesses. This will have a special focus on mould related disease. Clinical practice referrals my Company receives revolve around exposure to indoor air dampness and mould, usually in the home. When this is prolonged, it can cause problems including: vocal cord irritation, rhinitis, cough, wheezing and recurrent infections of the upper and lower respiratory tract. Breathing in bioaerosols might also make existing asthma worse or bring on new asthma in children or adults. Many of the client and patient referrals we receive complain that their doctor does not truly understand their symptoms or may even consider some of these to be psychosomatic. Apart from focussing on fixing the building, what can you do to ‘get well’ if you’re mould affected? To this end, an important paper recently came out in the Journal: Alternative Therapies in June 2021. Salt therapy is put forward as a treatment for mould related illness. But what is it and why does it work? Salt therapy has developed into a holistic complementary therapy available in day spa-like environments based on observations made by a 19th century Polish doctor, Feliks Boczkowski. He noticed that salt mine workers had far fewer skin and respiratory health symptoms compared with other mine workers. Later during World War II in Germany, people who sheltered during bombing in salt caves were observed to experience relief from respiratory problems. Salt therapy exploits or mimics the microclimate of natural salt caves. People who spend time in these caves are take advantage of what is called Spleotherapy in the literature. This involves inhaling pure sodium chloride, NaCl along with other aerosolized elements including magnesium and calcium. A different version termed Halotherapy allows people to experience the benefits of salt therapy by spending time in a room that has salt coated walls and floor. Halotherapy may also use a generator (termed a halogenerator) to produce fine sodium chloride aerosols (1-5-micron diameter) which are aerosolized in the ‘salt room’. The salt easily penetrates into all parts of the respiratory tract and has antibacterial and anti-inflammatory properties. Salt particles also promote ciliary transport in the lung, allowing mucus plugs to be coughed up and it also reduces IgE inflammatory levels. Doctors have used salt therapy in the treatment of allergic rhinitis, asthma, COPD, bronchitis for cystic fibrosis and for viral bronchiolitis. There is even research support linking the use of salting of face masks to neutralize the SARS-CoV-2 virus in an effort to combat COVID-19. Salt therapy is known to be 100% natural and it's generally safe because it improves the clearance of particulates from the lung, often provides relief from coughing and reduces bronchial inflammation. It’s easily experienced by spending time in a salt room (30-60 min. sessions) repeated 10 to 20 times. If your home might be mouldy, then consider salt your new ally for health promotion. REFERENCES: Wasik AA, Tuuminen T. Salt Therapy as a Complementary Method for the Treatment of Respiratory Tract Diseases, With a Focus on Mold-Related Illness. Altern Ther Health Med. 2021 Oct;27(S1):223-239. PMID: 34726628. https://pubmed.ncbi.nlm.nih.gov/34726628/ Preeti Shah. (2019). A Review of Salt Therapy as a Complementary Treatment for Enhancing Respiratory Wellness and Skin Ailment. The Indian Practitioner, 72(5), 28-32. Retrieved from https://articles.theindianpractitioner.com/index.php/tip/article/view/34 Tuuminen T. Dampness and Mold Hypersensitivity Syndrome, or Mold-related Illness, Has Become Highly Politicized and Downplayed in Finland. Alternative Therapies in Health and Medicine. 2021 May;27(3):59-64. PMID: 33882029. Rubino, I., Oh, E., Han, S. et al. Salt coatings functionalize inert membranes into high-performing filters against infectious respiratory diseases. Sci Rep 10, 13875 (2020). https://doi.org/10.1038/s41598-020-70623-9 #mould #salt #halotherapy
Salt Therapy and Mould Illness
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- Many high-income countries including Australia have been playing vaccine roulette with our public health. Lockdowns and personal restrictions have been applied while institutions wait for the vaccine effect to kick in. - I’m not here to question the principle of immunisation that prevents disease transmission and infection which in many areas of application is effective. BUT - The problem is that the majority narrative for vaccine safety has not adequately looked at complementary strategies like: other treatments, strengthening the health system, non-pharmaceutical prevention, promotion of safe infection control practices and perhaps above all, a healthy lifestyle. - This talk will look at another complementary strategy and is a follow-on from my OpEd paper and talk on the potential benefits of nicotine against COVID infection. - A comment on this video suggested I should make a video on some outstanding University of Michigan research just published at the end of August 2021. I was curious and looked into this. WOW…important research and I want to bring you an overview of this and how you can take advantage of this yourselves. - Despite vaccination there is a need for antiviral compounds that can (i) control local outbreaks, (ii) be used for home-based management and (iii) be used for immunocompromised people for whom vaccination is not suitable and this is required to (iv) slow the spread of variants of concern that may escape vaccine neutralisation. - Repurposing already FDA-approved drugs is a good place to start to look for drugs that could be deployed as treatments for COVID-19. - The U. Of Michigan research has developed a way to quantitatively screen actual SARS-CoV-2 infection in human cells and screen for drugs that show in vitro antiviral activity. - The scientists screened 1425 drugs and found 17 that inhibited coronavirus infection. - I’m focussing on the top #1 = Lactoferrin (bovine lactoferrin) - They screened the drugs using African Green monkey kidney cells called Vero E6 as well as several cancer cell lines to see how the virus and drugs worked together. This provided additional insight into the impact of underlying co-morbidity and cancer treatment protocols on infection status. - We’re going to only focus on coronavirus infection in normal healthy cells in this talk. - They screened the 1425 FDA-approved compounds at 5 concentrations and looked for antiviral activity. - The best performing drug was a nutraceutical called Lactoferrin. This is a protein found in colostrum and milk. It was able to block SARS-CoV-2 replication at the entry level. - It was also found to be effective as a post-entry inhibitor - They also found that a common asthma inhaler (ipratropium bromide) used in the treatment of asthma and COPD curbed infection and reduced severe bronchial symptoms. - How does lactoferrin work? It controls the up-regulation and down-regulation of the immune response. - It decreases the production of IL-6 which is produced by the white-bloc cell macrophages which respond to infection. By reducing the levels of IL-6 this can reduce the cytokine storm effect. - Lactoferrin is known to be a safe nutritional supplement anyone can buy online from the pharmacy. - Lactoferrin is also found in cheeses, specially hard cheese like Swiss cheese. - Lactoferrin is cheap, widely available and according to research can be rapidly deployed for both prophylaxis and management of COVID-19 - Lactoferrin is known to be: anti-microbial, antifungal, antiviral, and anti-parasitic. It possesses an anti-inflammatory, immunomodulatory, and anti-cancer molecule. It is also a fundamental regulator of cellular iron homeostasis. - In healthy condition, Lf concentration in blood is rather variable yet always relatively low. - To use lactoferrin for it’s anti-inflammatory and anti-cancer benefit, higher concentrations are needed to be effective, therefore, exogenous lactoferrin must be administered in vivo to overcome this drawback - Nutraceuticals like lactoferrin are natural compounds having potent antiviral activity, anti-inflammatory and anti-microbial activity and anti-cancer activity - A great candidate for combination therapies Mirabelli, C., Wotring, J., Zhang, C., McCarty, S., Fursmidt, R., Pretto, C., Qiao, Y., Zhang, Y., Frum, T., Kadambi, N., Amin, A., O’Meara, T., Spence, J., Huang, J., Alysandratos, K., Kotton, D., Handelman, S., Wobus, C., Weatherwax, K., Mashour, G., O’Meara, M., Chinnaiyan, A. and Sexton, J., 2021. Morphological cell profiling of SARS-CoV-2 infection identifies drug repurposing candidates for COVID-19. Proceedings of the National Academy of Sciences, 118(36), p.e2105815118. #lactoferrin #inflammation #covid
Lactoferrin and Covid - What You Need To Know
YouTube Video UEx6c1l0WHg1bVZ0dGptQUJGVDNEN1dyT3FPeGhfTGJ3OC4zMDg5MkQ5MEVDMEM1NTg2
Black mould is a common term used to describe home infestations with mould. It’s use is linked with an expectation of toxicity. In this week's Livestream, I report on the first case of Black mould sinusitis and how this was linked with immunocompromised status. Did you know that 17% of insurance claims at a major Australian insurer were water damage related? Did you know that in the USA, approximately 3.6% of the population are immunocompromised? Do you see a pattern here? Damp housing + immune compromise = opportunity for Black mould and other moulds to become BIG problems! Learn more on this Livestream or watch later or listen to the podcast later. REFERENCES: Semis M, Dadwal SS, Tegtmeier BR, Wilczynski SP, Ito JI, Kalkum M. First Case of Invasive Stachybotrys Sinusitis [published online ahead of print, 2020 Mar 10]. Clin Infect Dis. 2020;ciaa231. doi:10.1093/cid/ciaa231 Flappan SM, Portnoy J, Jones P, Barnes C. Infant pulmonary hemorrhage in a suburban home with water damage and mold (Stachybotrys atra). Environ Health Perspect. 1999;107(11):927-930. doi:10.1289/ehp.99107927 Massoud Mahmoudi & M. Eric Gershwin (2000) Sick Building Syndrome. III. Stachybotrys chartarum, Journal of Asthma, 37:2, 191-198, DOI: 10.3109/02770900009055442 Shinya Y, Miyawaki S, Nakatomi H, et al. Recurrent cerebral aneurysm formation and rupture within a short period due to invasive aspergillosis of the nasal sinus; pathological analysis of the catastrophic clinical course. Int J Clin Exp Pathol. 2015;8(10):13510-13522. Published 2015 Oct 1. Identifying a fungal signature of moisture damage in buildings: targeted and untargeted approaches with mycobiome data. Rachel I. Adams, Iman Sylvain, Michal P. Spilak, John W. Taylor, Michael S. Waring, Mark J. Mendell. Applied and Environmental Microbiology Jun 2020, AEM.01047-20; DOI: 10.1128/AEM.01047-20 Khan L. The growing number of immunocompromised - Bulletin of the Atomic Scientists. Bulletin of the Atomic Scientists. https://thebulletin.org/2008/01/the-growing-number-of-immunocompromised/. Published 2008. Accessed August 9, 2020. #mould #blackmould #stachybotrys
Facts About Black Mould and Fungal Sinusitis
YouTube Video UEx6c1l0WHg1bVZ0dGptQUJGVDNEN1dyT3FPeGhfTGJ3OC41MjE1MkI0OTQ2QzJGNzNG
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